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From a molecular point of view, the necroptosis pathway is initiated by auto- and trans-phosphorylation of RIPK1 and RIPK3 at Ser-227, resulting in stimulation of their kinase activity and assembly of a heterodimer protein complex termed necrosome via their RIPK homotypic interaction motif domains (Fulda, 2013

MTHFR C677T or A1298C variants reduce methylenetetrahydrofolate reductase activity, impairing the conversion of homocysteine to methionine and increasing homocysteine accumulation

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Ozbek L, Abdel-Rahman SM, Unlu S, Guldan M, Copur S, Burlacu A, et al