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Direct comparison of ML226 treatment to lentiviral ABHD11 sgRNA depletion demonstrated that both pharmacological and genetic inhibition of ABHD11 reduced functional lipoylation on the E2 subunit of the OGDHc (dihydrolipoamide succinyltransferase (DLST), referred to as the OGDHc-E2) in preference to the E2 subunit of PDHc (dihydrolipoamide acetyltransferase (DLAT), referred to as the PDHc-E2

In bone defects, application of P-15-containing bone substitutes increased the rate of bone growth compared to the defects left empty or filled with bone substitute alone [120, 121, 123, 124, 132, 138]

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