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A primary mechanism is its role as a redox agent: MB can accept electrons and undergo reduction to leucomethylene blue, and in doing so it helps convert oxidized hemoglobin (methemoglobin, Fe) back to functional hemoglobin (Fe)

A further randomized, placebo-controlled trial by Bor-Jen Lee et al

On the other hand, there is a possibility that TAT-DAT NT, which main action is to disrupt the D2R-DAT interaction, may affect phosphorylation of DAT and other DAT-associated protein interactions

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