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Anticonvulsants, antibiotics, antivirals, anti-gout, and anti-GERD treatments can increase the toxicity of an acetaminophen dose by induction of CYP enzymes, depletion of glutathione stores, or saturation of other liver detoxification systems (Amar 2007)
Individual results vary, and some patients may lose less or more than the averages reported in clinical research
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To determine whether SLC25A39 transport function is essential for GSH import into mitochondria, we generated a homology model of SLC25A39 based on that of the bovine ADP/ATP transporter 19 that facilitated the identification of three potential substrate binding residues, K329, M133 and D226 (Fig
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