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The expression of the -synuclein mutant A53T induces CMA dysfunction, which is mediated by the expression of -synuclein protofibrils [63]

Correcting the metabolic phenotype may lead to more favorable responsiveness and cumulative pregnancy outcomes in subsequent cycles, though prospective RCT validation is still required (170, 197)

Every hair follicle requires constant nutrient supply through tiny blood vessels called capillaries

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